https://ijvtpr.com/index.php/IJVTPR/article/view/66
In the main body of our text, we present 26 cases of Creuzfeldt-Jacob Disease, alldiagnosed in 2021 with the first symptoms appearing within an average of 11.38 days after a Pfizer, Moderna,or AstraZeneca COVID-19 injection. Because the causal progression, theetiopathogenesis, of these atypical and new cases of human prion disease—cases of what is apparently a totally new form ofrapidly developing Creuzfeldt-Jacob Disease—we focus on the chronology of the symptomatic development. We consider itfrom an anamnestic point of view —one in which we compare the typical development of pre-COVID cases of Creuzfeldt-Jacob Disease to the extremely accelerateddevelopment of similar symptoms in the 26 cases under examination. By such an approach, we hope to work out the etiopathogenesis critical to understanding this newand much more rapidly developing human prion disease. By recalling the sequential pathway of that the formerly subacute and slowly developing disease followed in the past, and by comparing it with this new, extremely acute, rapidly developing prion disease—one following closely usually after two of the COVID-19 injections—we believe it is correcttoinfer that the injections caused the disease in these 26 cases
The clinical facts reportedin this article confirm anunnatural, injection related,new form of Creutzfeldt-Jacob Disease. Thestereotypic post-vaccine symptoms appear almost immediately, or very soon after a jab, usually the second one,suggesting that the first injection is a potentiating event, all of which arefollowed by lightening-fast development of the disease progressing to its final conclusionin death.
https://www.mdpi.com/2076-393X/10/10/1651
However, histopathological analyses of the brain uncovered previously unsuspected findings, including acute vasculitis (predominantly lymphocytic) as well as multifocal necrotizing encephalitis of unknown etiology with pronounced inflammation including glial and lymphocytic reaction. In the heart, signs of chronic cardiomyopathy as well as mild acute lympho-histiocytic myocarditis and vasculitis were present. Although there was no history of COVID-19 for this patient, immunohistochemistry for SARS-CoV-2 antigens (spike and nucleocapsid proteins) was performed. Surprisingly, only spike protein but no nucleocapsid protein could be detected within the foci of inflammation in both the brain and the heart, particularly in the endothelial cells of small blood vessels. Since no nucleocapsid protein could be detected, the presence of spike protein must be ascribed to vaccination rather than to viral infection. The findings corroborate previous reports of encephalitis and myocarditis caused by gene-based COVID-19 vaccines.
Zakładki